CCM 3P — Cabinet Conseil en Micronutrition
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Micronutritional management of overweight and obesity: Part 2, practical applications

You have mastered the theory of the micronutritional management of overweight and obesity, but one question remains once the patient is sitting in front of you: concretely, where do I start and how do I bring all this information together? This practical part opens the doors of a real consultation, step by step, to…

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6 lessons · Lifetime access · Online

These trainings are delivered in French. This page is translated for your convenience; the course videos and materials remain in French.

Overview

You have mastered the theory of the micronutritional management of overweight and obesity, but one question remains once the patient is sitting in front of you: concretely, where do I start and how do I bring all this information together? This practical part opens the doors of a real consultation, step by step, to turn your knowledge into personalised protocols applicable from the very next day.

What you will learn

  • Build a relevant and prioritised functional biology assessment (from screening for metabolic syndrome to the calculation of HOMA and QUICKI, including micronutrient deficiencies, the thyroid, the digestive, mitochondrial and neurotransmitter profile and salivary cortisol)
  • Set up supplementation in two stages: a baseline supplementation based on the clinical picture, then a personalised supplementation reading the biological results
  • Explain simply to your patient the central role of insulin, lipogenesis and lipolysis, and de-demonise lipids using educational metaphors
  • Offer and adjust the two main dietary levers: carbohydrate modulation (low carb, glycaemic loads, net carbs, ketogenic diet) and dietary restriction windows (intermittent fasting)
  • Support the keto-adaptation phase and anticipate the keto flu with concrete strategies (citrates and bicarbonates, salt, bone broth, medium-chain triglycerides)
  • Deconstruct patients' frequent beliefs about fasting and adopt a coaching stance to encourage long-term adherence

On the programme

  • The use of functional biology: prescribing individual markers or an investigation panel, screening for metabolic syndrome, assessing insulin resistance and looking for the associated metabolic blocks (thyroid, gut, adrenals, mitochondria, neurotransmitters)
  • The commented reading of a fatty acid profile and the interpretation of the main digestive markers (LBP and metabolic endotoxaemia, candidiasis, microbiota)
  • The supplementation strategy, from baseline supplementation to adjustment according to the degree of insulin resistance (chromium, magnesium, berberine, antioxidants, support of hepatic detoxification, and more)
  • The dietary models explained to the patient: low carb and glycaemic loads, ketogenic diet and nutritional ketosis, food pyramid, distinction between healthy keto and dirty keto
  • Intermittent fasting and its different forms, from the restriction window to the one-meal-a-day, with the deconstruction of misconceptions
  • The practical consultation tools: weekly planners, recipes, shopping lists, protein portion calculation, and the organisation of follow-up (assessment consultation, interpretation, intermediate psychonutrition sessions)

Who it is for

This training is for healthcare professionals, doctors, pharmacists, naturopaths and micronutrition therapists, who see patients with overweight or obesity and wish to move from theory to a structured consultation practice.

At the end of the training

You will be able to conduct a complete consultation, prescribe and interpret a targeted functional biology assessment, then build a supplementation and a dietary model adjusted to each patient's profile and degree of insulin resistance. You will know how to support the implementation of the model, anticipate difficulties and adapt your approach to the person's beliefs and lifestyle. You will thus have a reproducible consultation framework, while bringing your own personal touch to it.

Programme

Total video duration: 6 h 21 min

CCM SURPOB MICRONUT PRATIQUE
  • Quiz de début de formation
  • VideoPDFPDFLeçon 11 h 47 min
  • VideoLeçon 21 h 29 min
  • VideoLeçon 31 h 28 min
  • VideoLeçon 41 h 37 min
  • Quiz de fin de formation

Video VideoAudio AudioPDFPDF PDF handout

Frequently asked questions

Why is intermittent fasting often easier to set up than the ketogenic diet?
Intermittent fasting requires no knowledge of macronutrients or meal planning: you simply do not eat during a defined window. It reduces the budget and time spent preparing meals, and imposes no forbidden foods, which limits frustration. It is a model particularly well suited to busy professionals and single people without evening family constraints.
How should the menus for the first weeks be concretely structured for a patient starting the low-carb diet?
Dr Anne Lucas, PharmD, recommends giving the patient a pre-filled weekly planner with example plates and recipes (homemade granola, zoodles, low-carb banana bread, fat bombs). Anticipation and organization are presented as the key to success, since this model differs greatly from typical French eating habits. It is advisable to select recipes in advance rather than leaving the patient to search alone on websites that are often in English.
What essential biological criteria should be checked first-line in an overweight or obese patient?
Dr Anne Lucas, PharmD, recommends starting with a standard panel to assess metabolic syndrome (waist circumference, HDL, triglycerides, blood glucose, blood pressure), then measuring fasting glucose and insulin to calculate HOMA and QUICKI. Chromium is checked systematically, as are vitamin D, erythrocyte magnesium, and LBP to detect metabolic endotoxemia. These few markers already provide solid clinical direction before going further into functional biology.
How can it be determined whether a patient should follow a simple low-carb diet or a full ketogenic diet?
Dr Anne Lucas, PharmD, recommends cross-referencing two clinical indicators: the HOMA score (insulin resistance) and the state of metabolic syndrome (waist circumference, lipid panel). The higher the HOMA, the stricter the model should be. It is also necessary to assess the absence of psychological blockage or severe cognitive restriction, which would contraindicate the keto diet.
How do you concretely build the meal timeline with a patient to introduce intermittent fasting?
Dr Anne Lucas, PharmD, suggests drawing a 24 hour timeline by hand and asking the patient to place their usual meals on it, referring to "meal 1, 2, 3" rather than breakfast/lunch/dinner. You then identify their current insulin-free period (often 8-10 hours) and their insulin-dominant period, then present the 16-8 target model to measure the gap and negotiate progressive adjustments.
What pre-analytical precautions are essential to obtain a reliable HOMA and avoid false negatives?
The patient must have eaten at least 30 to 50 grams of carbohydrates at the previous evening's meal, observe a 10-hour fast (12 hours maximum), and not have fasted the evening before. Without these conditions, glucose and insulin are artificially low and HOMA underestimates insulin resistance. The same rule applies to the candidiasis test (intestinal microbiota, MI) to avoid false negatives.
Who should absolutely not be offered a strict low-carbohydrate model?
Patients presenting significant cognitive restriction or an eating disorder should be excluded from the ketogenic diet: imposing the suppression of large food categories worsens hypercontrol and generates more compulsions. In this case, Dr Anne Lucas, PharmD, recommends working solely on psychonutrition.
What is "keto flu" and how do you prevent it?
Keto flu refers to the set of symptoms (headaches, intense fatigue, nausea, hypotension) that appear during the metabolic switch, when the body shifts from glucose (glycogen stores) to ketone bodies produced from fat. It is mainly linked to the electrolyte imbalance caused by the drop in glycogen. To limit it, Dr Anne Lucas, PharmD, recommends hydration rich in minerals, well-salted bone broth, two teaspoons of table salt per day, bicarbonate and citrate supplementation, and avoiding intense physical activity during this period.
How do you calculate and adjust protein intake during the keto-adaptation phase?
Dr Anne Lucas, PharmD, uses the Hume formula to calculate the ideal amount of protein based on current weight and target weight. During keto-adaptation, she recommends increasing this intake by 10 to 20 grams above the norm, to avoid proteolysis linked to gluconeogenesis. She recommends working in 20 gram protein portions (2-3 eggs, 150 g of fish, 100 g of meat, 60 g of cheese, etc.) and spreading intake across three main meals.
How should the DMI (intestinal microbiota panel) be interpreted to conclude whether intestinal candidiasis is present?
The candidiasis marker in the MI panel is D-arabinitol: an elevated level indicates candidiasis, independently of the D/L ratio. If the D/L ratio is normal despite an elevated D-arabinitol, this indicates a good eubiotic fermentation flora, which is rather a favorable sign. On the other hand, elevated arabinose alone, without an elevation of D-arabinitol, is not sufficient to conclude candidiasis and may simply reflect a high intake of apples or pears.
What is the difference between nutritional ketosis and therapeutic ketosis?
Therapeutic ketosis requires permanently staying under 20 g of carbohydrates per day and is reserved for precise medical indications. It has notably been validated in the treatment of drug-resistant epilepsy. Studies are currently underway for type 2 diabetes, Alzheimer's disease, Parkinson's disease, certain chronic inflammatory diseases, and certain cancers. Nutritional ketosis is more flexible: the tolerated amount of carbohydrates varies from 0 to 100 g depending on the patient's degree of insulin resistance and level of physical activity.
Which fasting window should be proposed first, and how should it progress if insufficient?
The base model is 16-8 (16 hours of fasting, 8 hours of food intake). If weight loss stalls, you progressively move to 17-7, 18-6, up to 20-4 (warrior fast) or OMAD (one meal a day). In cases of significant insulin resistance, eating in the first part of the day (8am-4pm option) is preferable to a later window (12pm-8pm) to limit nighttime insulin secretion.
What message should be given about lipids so as not to reinforce the patient's lipophobia?Full answer inside the training →
How does Dr Anne Lucas, PharmD, explain the keto-adaptation phase, and how long does it last?Full answer inside the training →
Why doesn't Dr Anne Lucas, PharmD, systematically order the urinary neurotransmitter panel, and when should it be considered?Full answer inside the training →
Can you lose weight with intermittent fasting alone, without reducing carbohydrates?Full answer inside the training →
Which drinks are allowed during the fasting period?Full answer inside the training →
How do you calculate the appropriate protein intake for an overweight patient?Full answer inside the training →
What baseline supplementation does Dr Anne Lucas, PharmD, put in place from the very first consultation, even before biological results are available?Full answer inside the training →
How do you progressively set up intermittent fasting after the keto-adaptation phase?Full answer inside the training →
How should supplementation be adjusted based on the HOMA result at the second consultation?Full answer inside the training →
How do you respond to the objection "fasting triggers famine mode"?Full answer inside the training →
How do you handle a patient who plateaus despite a 16-8 fast and under 50 g of carbohydrates per day?Full answer inside the training →
Can an excess of lipids block weight loss on a ketogenic diet?Full answer inside the training →
What micronutritional supplementation should be introduced from the first consultation, before lab results?Full answer inside the training →
Why does intermittent fasting not cause hypoglycemia in these patients?Full answer inside the training →
What are "glucinettes" and why are they preferable to glycemic loads for ketogenic follow-up?Full answer inside the training →
What metaphor is used to simply explain to the patient why carbohydrates cause weight gain but fats don't?Full answer inside the training →
How do you help the patient not feel deprived when giving up grains and bread?Full answer inside the training →
What are the two dietary models proposed to activate lipolysis and trigger the metabolic switch?Full answer inside the training →
How should the disappearance of ketone bodies in urine after a few weeks of ketosis be interpreted?Full answer inside the training →
Why is it necessary to provide direct EPA/DHA omega-3s rather than flaxseed or rapeseed oil in an obese patient?Full answer inside the training →
Why is it important to measure free T3 in 24-hour urine in addition to the blood thyroid panel?Full answer inside the training →
How do you incorporate legumes into a ketogenic diet without leaving nutritional ketosis?Full answer inside the training →
How do you reintroduce carbohydrates during the maintenance phase while maintaining nutritional ketosis?Full answer inside the training →
How should follow-up consultations be scheduled for a patient losing weight?Full answer inside the training →
What simple system does Dr Anne Lucas, PharmD, use to help the patient manage carbohydrate portions?Full answer inside the training →
How do you treat candidiasis and insulin resistance simultaneously in the same patient?Full answer inside the training →
How should functional biology panels be presented to the patient so they can choose according to their budget without feeling pressured?Full answer inside the training →
In which cases should the ketogenic model not be offered, and what alternative should be considered?Full answer inside the training →

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